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Ramos-Jacques at Edit Academy for her assistance in editing and proofreading the manuscript

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Intermittent dosing of rapamycin has been proposed as a means to prevent unwanted mTORC2 inhibition and the resulting metabolic defects in vivo (Arriola Apelo et al., 2016)

This challenge is exemplified by the early clinical setbacks of ES, which failed to improve overall survival, underscoring the critical need for precise patient stratification based on biomarkers such as mitochondrial metabolic capacity

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Repeated dosing of tyramine will result in (a form of rapid onset tolerance) (Figure 1)