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Beyond these established factors, emerging evidence suggests that the disruption of copper homeostasis and the induction of cuproptosis are critical integrative mechanisms that potentially link systemic inflammatory-metabolic stress to direct cardiomyocyte death [174]

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Treatment with 5% (w/v) ZIL also did not affect haemoglobin production, as evidenced by o -dianisidine staining, confirming the lack of effect of ZIL treatment on erythropoiesis (Fig

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