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The cell line was further stably transfected with receptor modifying protein 3 (RAMP3) thus generating the human amylin 3 (a) receptor (hAMY 3(a) R)

Consistent with this, Persoon and colleagues [95] found that the inactivation of all Rab3 genes in neurons resulted in the ablation of secretory granule fusion that was restored following the re-expression of Rab3a, suggesting a key role for Rab3a in the fusion process

Research shows Dihexa treatment increases expression of synaptophysin and other synaptic markers even in disease models characterized by synaptic degeneration

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Similar content being viewed by others Introduction Dementia is the most common neurodegenerative disease from which the Alzheimers disease (AD) is the most common among the elderly