Intercellular antioxidant support
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46 : 419430 Pettegrew JW, Minshew NJ, Cohen MM, Kopp SJ, Glonek T
This may also explain the loss of contractile function during metabolic inhibition (Figures 1, 2), where the Kir6.2/SUR2A channels, the well-established canonical cardiac K ATP channel subunits, would also be open (Brennan et al., 2024
10.1172/jci94601 J Clin Invest
J Appl Anim Res
It was Japanese scientists who first began to study it as a means of treating stomach diseases, including gastritis and ulcers
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