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LOS suppressed these AngII-mediated responses, whereas the AngII-independent upregulation of EMT-related proteins and the enhancement of mitochondrial energy metabolism by AngII in AGTR1-overexpressing cells remained unaffected

plantarum LP115 stimulated DAO release in vitro, human clinical trials are still needed before making firm recommendations

PMID 3663288

2013;11(3):28692 ( quiz e24 )

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322 By understanding these molecular mechanisms, personalized training and rehabilitation programs can be designed to address specific genetic predispositions and optimize injury prevention efforts