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doi: 10.1038/s41586-018-0004-7 58 MongaSPPediaditakisPMuleKStolzDBMichalopoulosGK

Endogenously oxidized mitochondrial DNA induces in vivo and in vitro inflammatory responses

Sulforaphane inhibits the interferon--induced expression of MIG, IP-10 and I-TAC in INS-1 pancreatic -cells through the downregulation of IRF-1, STAT-1 and PKB

However, due to the toxicity of mitochondrial inhibitors and the absence of experimental animal models, our understanding of the contribution of excessive mitochondrial ROS to in vivo disease is limited

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Results: 3035% increase in whole blood glutathione levels Improved lymphocyte and erythrocyte glutathione status Decreased oxidative stress markers Enhanced natural killer (NK) cell function 2