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Excess iron in these cells promotes oxidative stress and lipid peroxidation, creating ideal conditions for ferroptotic cell death

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Studies have explored the role of proteins and mutated genes other than ATP7B as contributors to the WD phenotype

In addition, according to Herbert et al

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The administration of polysaccharides derived from HEM has been shown to inhibit the reduction of SOD enzyme activity and improve the free radical scavenging ability of Caco-2 cells
