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found that exogenous -KG mimics glutaminase-mediated ferroptosis, while using a transaminase inhibitor, amino-oxyacetate (AOA), reverses ferroptosis [85, 86]

F-actin provides the force necessary for the formation of nascent protrusions and modifications in spine shape, such that actin polymerization/depolymerization is the main determinant of spine structural dynamics

reports research grant support, on behalf of their institution, from Novo Nordisk and advisory/consultancy fees from Boehringer Ingelheim, Eli Lilly & Company, Gila Therapeutics Inc, GLW-01, International Medical Press, Novo Nordisk, Pfizer and ViiV

In a broader context, our findings suggest that non-canonical pathways of Cu uptake may be activated under certain physiological and pathological conditions

To induce PC12 differentiation, NGF (50 ng/ml