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The genotype and treatment associated increase of 53BP1 + glial cells agreed with RNA-seq results and suggested a perturbed DNA repair response in APP/PS1 animals that may contribute to the higher vulnerability of OL to demyelination (21)

Q., Siegel, J

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Instead, we hypothesized that this bacterium has to rely on other sources for its energy production

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Saugstad 98 in 2005 talked about the oxygen radical disease of the newborn: oxidative stress may involve different organs, often simultaneously, giving rise to different signs and different clinical manifestations
