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Glutathione S-transferase p elicits protection against H2O2-induced cell death via coordinated regulation of stress kinases

Hoke, T

It has been shown that administration of -glutamylcysteine (GGC) increases cellular levels of GSH, circumventing the regulation of GSH biosynthesis by providing the limiting substrate (Nakamura et al., 2012)

Inhibition of neuroinflammation Neuroinflammation is a defense response of the CNS against biological, physical, chemical, and other damage factors

Serum-free insulin-like growth factor I correlates with clearance in patients with chronic renal failure