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Furthermore, excessive zinc disrupts zinc finger protein function by saturating binding sites, leading to protein misfolding and loss of DNA-binding specificity, which may further dysregulate immune gene expression

Loss of PRMT7 in the heart reduces symmetrical dimethylation of -catenin, enhances Wnt--catenin signaling, and promotes myocardial hypertrophy

This was paralleled by the accumulation of glucose-6-phosphate (6P) in steady state GSH-deficient FoB (Supplementary Fig

10.1002/glia.20467 48 QuertemontE.ErikssonC