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The physiology of vitamin B-12 is a multistep process that is characterized by multiple trafficking events, associations and dissociations among several binding/carrier proteins, specific and multiligand receptor-mediated endocytosis, intracellular trafficking into different cellular compartments, and protein-specific controlled exit (1, 2)

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Although removal of these skin layers induces some superficial damage at the donor site, the remaining skin retains deeper components of thermoeffector appendages (e.g., secretory coils of sweat glands and reticular cutaneous arterioles) and their neural connections

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