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The potential mechanism by which CoQ10 improves the quality deterioration of aging-related oocytes is closely associated with its antioxidant properties, enhancement of mitochondrial function, promotion of autophagy, and inhibition of apoptosis

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While mitochondrial stress normally signals for peroxisomal biogenesis, this adaptive pathway is exhausted in chronic asthma, leading to peroxisomal depletion and a subsequent failure in redox and lipid homeostasis (103)

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