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In addition to the acquirement of drug-resistant mutation, the development of cancer stem cells also causes CML persistence and relapse from TKI therapy 12,13,14

reported that in this scenario, since FTH is activated downstream of NF-B, downregulation of this transcription factor in cutaneous T-cell lymphoma results in reduced expression of FTH, which leads to an increase in LIP, causing huge formation of ROS [174]