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In addition, ROS modifies the intracellular influx of calcium, resulting in interstitial oedema, haemoconcentration, and ischemia, as well as further production of ROS, creating a self-perpetuating cycle [47]

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Recent studies in melanoma have identified a protein involved in iron flux at mitochondrialysosome contact sites, revealing that organelle-to-organelle iron compartmentalization plays a role in ferroptosis regulation