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CrossRef Diabetic Kidney Disease: From Epidemiology to Clinical Perspectives Cheol Whee Park Diabetes & Metabolism Journal.2014
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As single-cell analyses confirm ferroptosis through ACSL4 upregulation and mitochondrial shrinkage [179, 180], the challenge remains to harness lipid metabolisms regenerative capacity while suppressing peroxidation cascades, thereby advancing targeted neural repair strategies
Driven by the hypothesis that Long COVID is fundamentally rooted in neuroinflammation and mitochondrial dysfunction, clinical trials have begun testing metabolic interventions
