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Thus, the increases in levels of ROS favor the aggregation of A peptides, which prolong the feedback of the oxidative cycle, as described in the hippocampus of AD patients [6,15,24]

CaMKII activity decays with a time constant of ~10 s, 100 times longer than the Ca 2+ transient, suggesting that CaMKII plays a role in prolonging Ca 2+ initiation signal in the spine

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The molecule incorporates a C20 fatty diacid moiety linked via a gamma-glutamic acid spacer, which binds to serum albumin and confers the extended 5-day half-life enabling once-weekly dosing

The upregulation of GPX2 leads to the clearance of H 2 O 2 , the inhibition of apoptosis, and the weakening of oxidative stress

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