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In cellular cultures, the ALC-induced decrease in oxidative stress occurred not only by exerting a protective effect on mitochondrial structure and functioning, thus making the electron transport chain less prone to electron leak and superoxide production, but also by stimulating the mechanisms of endogenous cellular antioxidants defense [112]

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AR-67, a DNA Topo-Isomerase I Inhibitor, Demonstrates Acceptable Tolerability and Preliminary Activity in a Phase II Trial of Patients with Myelodysplastic Syndrome (MDS) and Chronic Myelomonocytic Leukemia (CMML)