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Mitochondrial respiratory complex I dysfunction promotes tumorigenesis through ROS alteration and AKT activation

USA 114 , 66756684 (2017)

The therapeutic potential of NO supplementation to restore S-nitrosylation, re-regulate ER stress responses, and inhibit the oncogenic activity of MYCN presents a promising avenue for addressing the treatment resistance and aggressiveness of NEPC

However, its important to note that many other more common conditions, including taking certain medications, are more likely to be the culprit: for example, a lack of dietary intake, taking medications for acid reflux or diabetes or an autoimmune condition, she adds

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Lett appl microbial 46:507512 Lieber CS (2002) S-Adenosyl-L-methionine and alcoholic liver disease in animal models: implications for early intervention in human beings