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doi: 10.1258/ebm.2012.012157 151 KruegerESLloydTSTessemJS

TMAO can upregulate scavenger receptors, such as cluster of differentiation 36 (CD36) and scavenger receptor class A type 1 (SR-A1), and promote the formation of foam cells, leading to development and progression of atherosclerosis ( Atherosclerosis: TMAO Activates ERS The ER, formed in continuity with the outer membrane of the nuclear envelope, plays a key role as a central eukaryotic membranous organelle and is responsible for the synthesis, folding, and maturation of membrane and secreted proteins, lipid biosynthesis, and calcium storage ( via ERmitochondrial stress in vitro and aggravates aortic valve lesions in mice ( Thrombus Formation: TMAO Promotes Platelet Reactivity Normal platelet function is essential for hemostasis and maintenance of a closed circulatory system

Lastly, the interplay between Nf-B and Nrf2 pathways significantly influences the progression of long-term COVID neuropsychiatric complications [200-202], and astrocyte-neuron communication via the astroglial xc-system is crucial for synaptic glutamatergic regulation [203]

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The body stores 95% of carnitine in the heart and skeletal muscles