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The vital role of DA in neuroinflammation was also evidenced by the observation that excessive DA released into the synaptic cleft may stimulate regional microglia directly and trigger a neurotoxic signal cascade (Thomas et al., 2008)

In contrast, rapid-acting agents such as ketamine induce acute mitochondrial metabolic reprogramming and mitophagy engagement, highlighting that early correction of bioenergetic and redox disturbances may suffice to trigger downstream synaptic and behavioral recovery

Studies have shown varying bioavailability percentages for carnitine intake

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TMAO serves as a prognostic marker for survival in CKD patients, correlating with glomerular filtration rate, C-reactive protein, and cystatin C (137)