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Discussion According to our previous findings, plasma treatment caused a number of chemical modifications on GSH and GSSG in solution, giving a large number of (partly unidentified) S- and N-modified glutathione species as intermediates and final products (Fig

Blood 108, 25692577

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The choice between them affects stability and storage duration, not the calculation

DOI: 10.1016/C2014-0-00100-1 2

The systolic and diastolic defects are seen in the taurine deficit hearts that is linked to decreased sarcoplasmic reticular Ca 2+ ATPase activity, a modification arbitrated in part by decreased phospholamban phosphorylation [56]