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Secondly, fluid redistributions from the intravascular to the interstitial space are a consequence of diminished transmural pressure

FBXL5 deficiency in CD11c + myeloid cells promotes neutrophil infiltration during APAP-induced liver injury We next investigated how FBXL5 deficiency in CD11c + myeloid cells exacerbates APAP-induced liver injury

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This interaction is inhibited at low Ca 2+ concentration by the autoinhibitory domain (residues 595639) (Chen and Wu, 2000)

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Liao ST, Han C, Xu DQ, Fu XW, Wang JS, Kong LY
