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Bcl-2 acts as an anti-apoptotic protein, counteracting BAX activation to maintain this balance of programmed cell death [43]

Their liposomal glutathione stores the molecule within these vesicles, protecting it from breaking down
These modified compounds are then conjugated to polar compounds in phase II reactions

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However, when the acute inflammatory response is self-limited through NETs and AGGNets, residual immune stimulation can cause inflammation to transition from a reversible acute phase to a chronic remodeling phase if the crystals are not completely removed (137)

By supporting mitochondrial function, they ensure that cells efficiently convert nutrients into energy, promoting overall metabolic health
