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More than one way to die: apoptosis, necrosis and reactive oxygen damage

Ginsenoside Rg5 (G-Rb5) inhibited the activation of apoptotic pathway by increasing the expression of Bcl-2 and decreasing the level of Bax, and significantly reduced cisplatin-induced nephrotoxicity, oxidative stress and inflammation (Li et al., 2016

e , Western blot of HT1080 OE GFP-GPX4 WT after treatment with ferroptosis inducers with GPX4 and GAPDH antibodies

10.1155/2024/5594462 14 FaizaI

Altogether, our data corroborate the link between infection-induced impaired ketogenesis and dysfunctional CD4 + T cell immunity by showing that restoration of BHB supports the functionality of type 1 immune responses in viral infection

GSH functions as an enzyme cofactor for Grxs, which are low-molecular-weight redox enzymes that are also known as thiol transferase, to maintain cellular redox homeostasis, and also acts as the primary reductant of the disulfide bonds of oxidized proteins [15] (Figure 1)