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25 Furthermore, the Nrf2-Keap1 system is an evolutionarily conserved intracellular defense mechanism which plays a pivotal role in regulating antioxidant defenses

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The change in second messenger impacts further downstream effectors that ultimately lead to a cellular response

By further elucidating the pathogenic mechanism by which hepatic iron accumulation triggers ferroptosis through the c-Myc-Acsl4 regulatory axis, thereby accelerating the progression of metabolic-associated steatohepatitis (MASH), our research team identified a novel iron chelator, FOT1 (FerroTerminator1, formerly known as CN128), through drug screening, which can prevent and treat the onset and progression of MASH disease [117]

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