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The inhibition of ferroptosis as a treatment approach for CAC presents varying outcomes and warrants further investigations

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In NAFLD or MASLD, the liver undergoes a self-perpetuating cycle of IR and lipotoxicity, leading to the dysregulation of key metabolites involved in energy homeostasis [28]

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Blockade of glucagon receptor induces -cell hypersecretion by hyperaminoacidemia in mice

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