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Sucrose effectively enhances the postnebulization stability of mRNALNPs at 20C for 30 days, while LNPs without sucrose exhibit stability at 4C for 7 days and instability at 20C for 30 days [82]

While GSH has long been recognized as a key brain antioxidant, its role in neurodegenerative diseases is now being unraveled with mechanistic clarity: future research will likely focus on activating astrocytic Nrf2 signaling to enhance glutamate cysteine ligase (GCL)-mediated GSH synthesis, exploring the role of MCT1/2 transporters in neuronal GSH precursor supply, and dissecting how oxidative stress modulates microglial activation via pathways like NF-B-dependent proinflammatory cytokine secretion (Sun et al., 2022)

Figure 3 STW3-VI increases intracellular reduced glutathione in differentiated mouse hippocampal HT-22 neurons Cytoprotective enzymes and antioxidants are able to limit the cellular damage and thus, to control the toxicity of free radicals

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Improved recovery post-exercise and potential benefits in tissue repair [57,58]

The brain expresses 200-fold higher levels of this protein than most other cells (276)