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This can lead to apoptosis (cell death), mitochondrial dysfunction, oxidative stress, and ultimately, cognitive impairment

Mechanistic studies have revealed that altered expression of iron metabolism-related proteins (including GPX4, FTH1, TfR1, and HO-1), accumulation of lipid peroxidation products, and disruption of antioxidant defense systems (particularly the Nrf2/GPX4 pathway) are central to ferroptosis progression in cardiac tissue

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Lunn JS, Hefferan MP, et al

Eine parenterale Anwendung sollte deshalb klinisch berwacht werden, so eine narrative bersichtsarbeit aus dem Jahr 2025