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It has been determined that when glutamine depletion activates the energy stress AMPK pathway and inhibits mTORC1 activity, this results in the subsequent inhibition of the protein expression level of -TrCP, consequently leading to abnormal cell cycle progression and reduced proliferation [19]
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found that SLC38A2 was significantly upregulated as a compensatory transporter of glutamine when SLC1A5 was absent or inhibited in leukemia cells [28]
Habib, and S
Development of MDM2 degraders based on ligands derived from Ugi reactions: lessons and discoveries
