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[DOI] [PMC free article] [PubMed] [Google Scholar] 88.Jing L., Zhang H., Xiang Q., Hu H., Zhai C., Xu S., Tian H

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In contrast to mutations in SLC39A8, mutations in the Mn importer SLC39A14 lead to the accumulation of Mn in the brain and in various other extrahepatic tissues, which is characterized by neurological deficits, motor dysfunction, and gastrointestinal damage 48,49,50,51

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