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J Endocrinol (2004) 180(2):24755.10.1677/joe.0.1800247 224 WuSYangWDe LucaF

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Epigenetic modifications, including hypomethylation of ER and aromatase promoters, further sustain this estrogen-driven phenotype, with endocrine-disrupting chemicals (EDCs) such as dioxins and phthalates potentially exacerbating these epigenetic alterations ( Progesterone resistance, characterized by impaired progesterone receptor (PR) signaling despite bioavailable progesterone, perpetuates ectopic lesion survival and contributes to infertility ( The interplay of estrogen dominance and progesterone resistance disrupts endometrial homeostasis, directly impacting fertility

R., et al., Mucuna pruriens , A Possible Treatment for Depressive Disorders, Neurology International 16, no

Toxicol., 4: 808-818

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