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Mitochondrial dysfunction is increasingly recognized as a contributor to neuropathy, particularly in diabetic and chemotherapy-induced forms where mitochondrial damage in dorsal root ganglion neurons precedes and contributes to axonal degeneration

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Preclinical findings show measurable increases in microvessel density at BPC-157-treated wound sites compared to control groups within 7 to 14 days of administration

Enhanced MSC recruitment potentially increasing osteoblast and chondrocyte numbers at healing sites

emollient adjuncts or reduced application frequency often mitigate this effect

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