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Dujovne P

The Journal of Microbiology

One potential mechanism is the Nrf2-ARE pathway, which induces a phase 2 detoxification response that promotes disruption of Nrf2-Keap protein interactions, translocation of Nrf2 to the nucleus, and regulation of target gene expression through the ARE, enhancing cellular defense against oxidative damage (Sharma et al., 2010)

GKRS may be a viable and safe therapeutic alternative for patients with PRLomas who are either intolerant or resistant to DAs, thus males are more likely to be eligible for such treatment

[PubMed] [Google Scholar] 369.Shimomura Y, Suzuki M, Sugiyama S, Hanaki Y, Ozawa T

Clinical anti-tumor therapies that disrupt ROS homeostasis Conventional anti-tumor therapies, such as radiotherapy and chemotherapy, as well as emerging strategies such as photodynamic therapy, aim to deliberately increase ROS levels to selectively target and eliminate cancer cells