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Thats not a rat study
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doi: 10.3390/ijms23179588 23
Additionally, the extract negatively modulated GLI1 transcriptional activity by inhibiting SMO/PTCH1 molecules, subsequently regulating the intrinsic apoptotic signaling cascade [92]
however, understanding the cell-type-specific mechanisms of cellular dysfunction is a fundamental necessity to develop precision medicine strategies that could overcome these abnormalities and promote regeneration of spermatogenesis
181 SeibtT
