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In complex I, NADH is oxidized to produce electrons, which are then transferred to oxygen to generate O 2 [38]

Erastin exploits the defective mitophagy-driven ROS accumulation to circumvent the ferroptosis resistance characteristic of EESCs, thereby enhancing its therapeutic efficacy

This suggests that antibiotics and available chemotherapeutics do not supply effective treatment for such serious health problems

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Having only one time point in mouse trial makes the judgment more difficult