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These effects were reversible with L-carnitine treatment [16]

Carnitine is found almost exclusively (99%) intracellularly, existing either as free LC or as several species of acyl-carnitine esters, predominantly in the muscle and liver

This mechanism is complementary to other cognitive support strategies

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High TMAO levels in CKD patients have been associated with increased mortality and adverse cardiovascular outcomes, suggesting that TMAO could be a critical factor in the progression of CKD and its complications [72,73]

L-carnitine can be re-circulated once fatty acids enter the mitochondrial matrix, therefore only a small amount of L-carnitine is required in order to allow fatty acid to enter the mitochondria