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The release of damage-associated molecular patterns (DAMPs) and pro-inflammatory cytokines, such as TNF-, IL-1, and IL-6, from dying endothelial and immune cells further exacerbates systemic inflammation (Xiao et al., 2023

Crucially, this criterion applies equally well to existing molecules and to new (never synthesised) species, provided a sufficient degree of structural similarity can be identified among them

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While the first studies showing this were in mice [14], human studies have corroborated these findings [200], although its importance in relationship to traditional clearance pathways across the BBB and subarachnoidal granulations has been challenging to quantify