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doi: 10.3109/10428199509051709

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Moreover, ferroptosis inhibitors can effectively suppress Golgi apparatus stress by affecting its morphology and function, which further promotes the recovery of Golgi apparatus homeostasis[65]

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These findings led to the hypothesis that THRSP may mediate the T3-induced suppression of HCC progression and glucose metabolism by down-regulating ENO2 transcription and expression

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However, the results obtained demonstrated that even though the antioxidant inhibited GSH decrement and attenuated the inflammatory response, the skeletal muscle suffered for efficient recovery from the damage, suggesting that blunting the redox-mediated signaling pathways is not beneficial

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BZ is a centrally acting anticholinergic agent that was originally developed as a gastrointestinal antispasmodic but due to severe central nervous system effects was abandoned as a pharmaceutical

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Enhanced healing of ulcers in patients with diabetes by topical treatment with glycyllhistidylllysine copper

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