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Synergistic Cardioptotection by Tilianin and Syringin in Diabetic Cardiomyopathy Involves Interaction of TLR4/NF-b/nlrp3 and PGC1a/SIRT3 Pathways

Without adequate NAD+, Sirtuin activity falls and the cell's ability to repair itself degrades with it

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This mechanism follows a similar sequence of events that we found for the effect of AMPH on the DAT in DA neurons

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