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Emerging evidence indicates a mechanistic interplay between the Nrf2Keap1 axis and the NLRP3 inflammasome, wherein Nrf2 activation not only counteracts oxidative stress but also suppresses NLRP3-mediated inflammatory signaling

data curation, C.T

and logK4 2.22 [77]

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These progenitors exhibit pro-fibrotic and pro-inflammatory differentiation trajectories in response to joint injury
doi: 10.1111/j.1365-4632.2004.02027.x 27 ShajilEMBegumR