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383 Because mitochondrial and non-mitochondrial enzymes produce ROS during hyperoxia (70% O 2 ), the inability of mitochondrial antioxidants to reverse growth arrest in hyperoxia-induced senescence suggests that cytosolic ROS may assist growth arrest

Systematic post-market surveillance and long-term safety studies would address these knowledge gaps as clinical use potentially expands

However, only two pH values were evaluated, so no specific conclusion could be drawn from those studies [25]

In the meantime, access is workable but requires both a qualified prescriber and a compliant pharmacy

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78 Meanwhile, circ-Astn1 in ADSC-Exos instigated wound healing in diabetic mice via the miR-138-5p/SIRT1/FOXO1 axis