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doi: 10.1016/j.otohns.2010.04.266

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Another new compound, SU11274, that targets the c-Met ATP-binding site has also been reported and implicated in blocking of HGF-dependent c-Met activation ( In vivo experiments showed that daily administration of SU11274 in mice resulted in inhibition of tumor growth in xenografts

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Transient increases in hepatic transaminase activity have been reported with CaNa 2 EDTA, DMSA, and DMPS, but hepatic toxicity resolves with discontinuation of the medication