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These studies highlight the growing potential of gene therapy not only as a means to address the core mechanisms of AMD, such as neovascularization and inflammation, but also as a strategy to combat oxidative stress, one of the key drivers of retinal damage in dry AMD

8D), indicating that TMAO-induced Ca 2+ influx was required for ERK1/2 activation

J Neurochem 137:714729 Faul M, Xu L, Wald MM, Coronado VG, Traumatic brain injury in the United States: emergency department visits, hospitalizations and deaths 20022006

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doi: 10.3791/54879