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[DOI] [PubMed] [Google Scholar] 52.Mukherjee S., Goswami S., Dash S., Samanta D

[DOI] [PubMed] [Google Scholar] 67.Abdul Muneer PM, Alikunju S, Szlachetka AM, Haorah J

[3] and our observations [10, 11] may soon guide the development of novel and relevant clinical studies that aim to increase the knowledge of G6PD deficiency and explore the clinical relevance of the relationship of G6PD-deficient variants with the pathophysiology of CVD

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However, their activity decreases with aging, creating an imbalance between oxidative damage and repair mechanisms.12 Excessive accumulation of reactive oxygen species (ROS) within hair follicle melanocytes leads to cellular damage through lipid peroxidation, DNA strand breaks, mutations, and protein and enzyme denaturation

Indeed, one of the DKD features is the podocyte loss with consequent albuminuria (Pagtalunan et al., 1997