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nk kemoterapi gibi beden iin ykc etkiye sahip tedavilerde koruyucu mekanizmalara ihtiyacmz var
This is achieved through the redox modification of protein thiols and methionine mimicry in critical proteins, which can halt the cell cycle in the G1 phase ( Figure 3 )
Upregulation of the activity of Nrf2 and, subsequently, the antioxidant enzymes, could prevent the onset of DR complications in diabetic patients

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HF is an immediate target of CsA, and hypertrichosis may be the most intriguing and most common adverse effect of CsA 218

Nathan, N (May 11, 2011)