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It is widely believed that activation of the TH17 pathway by IL-23 is the predominant mechanism involved in the pathogenesis of psoriasis

This could possibly be carried out given its special interaction and modulatory effects with the nitric oxide (NO) system [43,44,45,77,78,79,80,81], thrombocytes [81,82,83], and many molecular pathways [36,37,43,44,45,46,47,48,49,50,51], particularly NO molecular pathways [43,44,45], which is a point also noticed in myotendinous junction recovery [13]

Moreover, it is notable that agonist E max values for receptor internalization matched those for -Arr recruitment for all agonists tested