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Additional investigation on the moderated expression of receptors and transporters during disease states could aid in better optimization of such approaches

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Interestingly, silencing of oct 3 transporter in mice attenuates both the MPP + release from astrocytes and the subsequent impairment of dopaminergic neurons, in which makes oct3 as an important molecular target for dopaminergic related pathologies [6,84]

This process includes a cascade of nuclear gene transcription to upregulate the expression of genes encoding specialized mitochondrial chaperones 164

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Taken together, these considerations foster the importance of individualizing LT4 replacement therapy, in order to align it not only with biochemical targets but also with the clinical context and physiological nuances of each patient (28)