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Mitochondrial dysfunction in preeclampsia is marked by abnormal mitochondrial morphology, impaired oxidative phosphorylation, and decreased activity of key electron transport chain proteins, especially in early-onset cases [109, 113]
That gap in data means no responsible recommendation can be made either way

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Protein degradation by the ubiquitinproteasome system Ubiquitin marks intracellular proteins for proteasomal degradation Hepatic protein turnover is highly regulated, which allows metabolic pathways to adapt to changing physiologic circumstances

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