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Correlation of the clinical severity with either VLCAD activity or C14/C16-acylcarnitine ratio was much less clear [124]
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and 3) down-regulating the expression of ABCA1 and ABCG1 in macrophages, potentially due to suppression of liver X receptor (LXR) activity by the TMA/FMO3/TMAO axis [4, 36, 37]
Age-related GSK3 overexpression drives podocyte senescence and glomerular aging
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